"Multiple Sclerosis, Diabetes, Parkinson's and Alzheimer's are not diseases. They are reactions to disease."
After two decades of studying fat embolism, I have uncovered a fundamental shift in our understanding of how plaque forms within the body.
Plaque formation is not merely a metabolic byproduct driven by cholesterol; it’s an active, reactive response to trauma—a “calcium ion crisis” within our cells and blood vessels. This crisis redefines conditions traditionally associated with plaque, such as atherosclerosis, Multiple Sclerosis, Parkinson’s, Alzheimer’s, diabetes, kidney stones, gallstones, arthritis, and more.
Here’s what I’ve found: trauma—whether chemical, physical, or environmental—triggers what I call a cellular “mini-bang.” Just as stars react to intense internal pressures by releasing elements in a cosmic burst, our cells respond to trauma by releasing calcium ions in a powerful, reactive cascade. These ions flood out of cellular stores like the sarcoplasmic or endoplasmic reticulum in direct proportion to the level of stress. The greater the trauma, the greater the number of calcium ions released, like cosmic building blocks mobilized to protect the body. Once released, these ions bind with LDL, riding on its “back” to sites of injury. Far beyond simply delivering cholesterol, this calcium-LDL complex drives both lipid deposition and rapid plaque calcification, transforming it into a rigid structure that serves as a protective but ultimately harmful response.
This discovery reframes our understanding of common conditions such as Atherosclerosis, Multiple Sclerosis, Parkinson’s, Alzheimer’s, diabetes, kidney stones, gallstones, arthritis, coronary artery disease and peripheral artery disease. The plaques and deposits observed in these conditions are the effect of trauma—the body’s innate immune response mobilizing fats, lipids, and plaques as a protective measure. What we traditionally label “autoimmune” is, in fact, innate immune—the body’s natural defense mechanism enlisting fats, lipids, and plaques to mitigate damage.
In fact, even at life’s end, the body continues its protective response. The final plaque formation—rigor mortis—represents one last flood of calcium, the body’s ultimate attempt to defend and preserve its structures.
The implications of this “calcium ion crisis” understanding are transformative, opening the door to a novel approach to treatment. I have a concrete idea for addressing this calcium-driven cascade and am actively seeking research partners and collaborators to further develop and implement this therapeutic breakthrough.
If you’re interested in joining forces to address this new frontier in cellular health, please reach out.
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